Controversial Claims Resurface: Was SARS-CoV-2 Engineered?

The debate surrounding the origin of the SARS-CoV-2 virus has been reignited by claims from Argentine neuropsychiatrist Gabriel de Erausquin, a member of an international research consortium. De Erausquin alleges that the virus, responsible for the global COVID-19 pandemic, was not of entirely natural origin, suggesting it was either “synthetic” or “semi-synthetic,” crafted within a Chinese laboratory.

This assertion echoes earlier reports, notably those from Chinese virologist Li Meng Yan, who initially raised concerns in Shanghai before seeking refuge in California, citing threats from the Beijing regime. While these claims remain contentious, they fuel ongoing investigations into the virus’s genesis.

Supporting Research and Biosecurity Concerns

De Erausquin further contends that research conducted in 2020 by teams in both the Netherlands and the united Kingdom independently corroborated the hypothesis of artificial manipulation of the SARS-cov-2 protein. These findings, if validated, would lend credence to the theory that the virus was initially conceived as part of a vaccine development project. Though, a lapse in biosecurity protocols at a Wuhan, China laboratory allegedly led to its accidental release into the wider population.

The hypothesis of the artificial origin of the virus was confirmed in 2020 by two research teams… which reached similar conclusions on the manipulation of the SARS-COV-2 protein.

While the specifics of the project’s funding and the researchers involved are reportedly accessible via the White House’s official website, the precise mechanism by which the virus began circulating remains unclear. this lack of clarity continues to fuel speculation and demands for a transparent and thorough investigation into the pandemic’s origins.

Long COVID and Alzheimer’s: A Troubling Connection?

Beyond the immediate impact of COVID-19,the long-term sequelae,commonly known as “Long COVID,” are becoming an increasing concern. De Erausquin highlights emerging research suggesting a potential genetic link between prolonged COVID-19 symptoms and Alzheimer’s disease (AD). This connection raises serious questions about the pandemic’s potential long-term neurological consequences.

Specifically, research indicates that individuals with a pre-existing genetic predisposition may be more susceptible to developing persistent symptoms following SARS-cov-2 infection, potentially leading to earlier manifestations of Alzheimer’s. This is particularly concerning given the already rising rates of dementia worldwide. According to the Alzheimer’s Association, in 2024, an estimated 6.9 million Americans aged 65 and older are living with Alzheimer’s. This number is projected to rise to nearly 13 million by 2050, underscoring the urgency of understanding any factors that could accelerate the onset of this debilitating disease.

Cognitive Impairment: A Tripling of Alzheimer’s-Like Symptoms

The Erausquin team’s research has revealed a disturbing trend: a tripling of symptoms consistent with the early stages of Alzheimer’s disease in individuals aged 60 to 70 with no prior history of cognitive decline following COVID-19 infection.These symptoms include:

• Persistent loss of smell (anosmia)
• Short-term memory loss
• Difficulty organizing daily tasks (impaired executive function)
• Difficulty recalling words or names

These findings underscore the need for continued research into the long-term neurological effects of COVID-19 and their potential association with neurodegenerative diseases like Alzheimer’s. Further investigation is crucial to determine the underlying mechanisms driving this connection and to develop strategies for mitigating the risk of long-term cognitive decline in post-COVID patients.

the Imperative of Biosecurity

The ongoing debate surrounding the origins of SARS-CoV-2, coupled with the emerging evidence linking long COVID to potential neurological complications, highlights the critical importance of robust biosecurity measures in laboratories handling potentially hazardous pathogens. The need to prevent future pandemics is paramount, and stringent protocols are essential to minimize the risk of accidental releases.